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影响因子: 1.352 5年影响因子: 3.347 SJR: 1.022 SNIP: 0.55 CiteScore™: 2.19

ISSN 打印: 1040-8401
ISSN 在线: 2162-6472

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购买问题 $142.00 卷 26, 2006 册 5

DOI: 10.1615/CritRevImmunol.v26.i5

内容列表:

Heavy-Chain-Only Antibody Expression and B-Cell Development in the Mouse
Marianne Bruggemann, Jennifer A. Smith, Michael J. Osborn, Daniel Corcos, Xiangang Zou, Viet Khong Nguyen, Serge Muyldermans
pages 377-390
DOI: 10.1615/CritRevImmunol.v26.i5.10
Structure and Function of Cas-L and Integrin-Mediated Signaling
Sachiko Seo, Motoshi Ichikawa, Mineo Kurokawa
pages 391-406
DOI: 10.1615/CritRevImmunol.v26.i5.20
Role of cAMP Phosphodiesterase 4 in Regulation of T-Cell Function
Elisa Bjorgo, Kjetil Tasken
pages 443-452
DOI: 10.1615/CritRevImmunol.v26.i5.40
The Role of NKT Cells in Animal Models of Autoimmune Hepatitis
Gunther Dennert, Fred Aswad
pages 453-474
DOI: 10.1615/CritRevImmunol.v26.i5.50

免疫学评论综述™

Model for the recruitment of inducer-specific TNF-α gene enhanceosome in T lymphocytes., Vol. 29, Iss.2, Viral Mechanisms Involved in the TranscriptionalCBP/p300 Regulation of Inflammat..... Model for the recruitment of inducer-specific TNF-α gene enhanceosome in T lymphocytes., Vol. 29, Iss.2, Viral Mechanisms Involved in the TranscriptionalCBP/p300 Regulation of Inflammat..... Viral Mechanisms Involved in the Transcriptional CBP/р300 Regulation of Inflammatory and Immune Responses
Activation of several B cells is necessary for exosome release.Vol. 29, Iss. 3, Exosome Release by Primary B Cells. Activation of several B cells is necessary for exosome release.Vol. 29, Iss. 3, Exosome Release by Primary B Cells. Exosome Release by Primary B Cells
Homology model of human GrM. Vol. 29, Iss. 4, The Biology of Granzyme M: A Serine Protease with Unique Features. Homology model of human GrM. Vol. 29, Iss. 4, The Biology of Granzyme M: A Serine Protease with Unique Features. Biology of Granzyme M: A Serine Protease with Unique Features
(A) Gap-junction formation, (B) A three-dimensional collagen matrix Vol. 30, Iss. 6, Endothelial Cells as Key Determinants of the Tumor Microenvironment. (A) Gap-junction formation, (B) A three-dimensional collagen matrix Vol. 30, Iss. 6, Endothelial Cells as Key Determinants of the Tumor Microenvironment. Endothelial Cells as Key Determinants of the Tumor Microenvironment: Interaction with Tumor Cells, Extracellular Matrix and Immune Killer Cells
Biphasic HCV RNA decline after IFN treatment of chronic hepatitis C. Vol. 30, Iss. 2, Treatment of Hepatitis C Virus Infection with Interferon and Small Molecule Direct Antivirals. Biphasic HCV RNA decline after IFN treatment of chronic hepatitis C. Vol. 30,  Iss. 2, Treatment of Hepatitis C Virus Infection with Interferon and Small Molecule Direct Antivirals. Treatment of Hepatitis C Virus Infection With Interferon and Small Molecule Direct Antivirals: Viral Kinetics and Modeling