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影响因子: 1.352 5年影响因子: 3.347 SJR: 0.657 SNIP: 0.55 CiteScore™: 2.19

ISSN 打印: 1040-8401
ISSN 在线: 2162-6472

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购买问题 $166.00 卷 29, 2009 册 2

DOI: 10.1615/CritRevImmunol.v29.i2

内容列表:

Integrin Function in T-Cell Homing to Lymphoid and Nonlymphoid Sites: Getting There and Staying There
Christopher C. DeNucci, Jason S. Mitchell, Yoji Shimizu
pages 87-109
DOI: 10.1615/CritRevImmunol.v29.i2.10
How Phagocytes Track Down and Respond to Apoptotic Cells
Helena Paidassi, Pascale Tacnet-Delorme, Gerard J. Arlaud, Philippe Frachet
pages 111-130
DOI: 10.1615/CritRevImmunol.v29.i2.20
Viral Mechanisms Involved in the Transcriptional CBP/р300 Regulation of Inflammatory and Immune Responses
Yolanda Revilla, Aitor G. Granja
pages 131-154
DOI: 10.1615/CritRevImmunol.v29.i2.30
Regulation of Ca2+ Signaling with Particular Focus on Mast Cells
Hong-Tao Ma, Michael A. Beaven
pages 155-186
DOI: 10.1615/CritRevImmunol.v29.i2.40

免疫学评论综述™

Model for the recruitment of inducer-specific TNF-α gene enhanceosome in T lymphocytes., Vol. 29, Iss.2, Viral Mechanisms Involved in the TranscriptionalCBP/p300 Regulation of Inflammat..... Model for the recruitment of inducer-specific TNF-α gene enhanceosome in T lymphocytes., Vol. 29, Iss.2, Viral Mechanisms Involved in the TranscriptionalCBP/p300 Regulation of Inflammat..... Viral Mechanisms Involved in the Transcriptional CBP/р300 Regulation of Inflammatory and Immune Responses
Activation of several B cells is necessary for exosome release.Vol. 29, Iss. 3, Exosome Release by Primary B Cells. Activation of several B cells is necessary for exosome release.Vol. 29, Iss. 3, Exosome Release by Primary B Cells. Exosome Release by Primary B Cells
Homology model of human GrM. Vol. 29, Iss. 4, The Biology of Granzyme M: A Serine Protease with Unique Features. Homology model of human GrM. Vol. 29, Iss. 4, The Biology of Granzyme M: A Serine Protease with Unique Features. Biology of Granzyme M: A Serine Protease with Unique Features
(A) Gap-junction formation, (B) A three-dimensional collagen matrix Vol. 30, Iss. 6, Endothelial Cells as Key Determinants of the Tumor Microenvironment. (A) Gap-junction formation, (B) A three-dimensional collagen matrix Vol. 30, Iss. 6, Endothelial Cells as Key Determinants of the Tumor Microenvironment. Endothelial Cells as Key Determinants of the Tumor Microenvironment: Interaction with Tumor Cells, Extracellular Matrix and Immune Killer Cells
Biphasic HCV RNA decline after IFN treatment of chronic hepatitis C. Vol. 30, Iss. 2, Treatment of Hepatitis C Virus Infection with Interferon and Small Molecule Direct Antivirals. Biphasic HCV RNA decline after IFN treatment of chronic hepatitis C. Vol. 30,  Iss. 2, Treatment of Hepatitis C Virus Infection with Interferon and Small Molecule Direct Antivirals. Treatment of Hepatitis C Virus Infection With Interferon and Small Molecule Direct Antivirals: Viral Kinetics and Modeling