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ISSN 打印: 1045-4403

ISSN 在线: 2162-6502

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 1.6 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.2 The Immediacy Index is the average number of times an article is cited in the year it is published. The journal Immediacy Index indicates how quickly articles in a journal are cited. Immediacy Index: 0.3 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00058 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.33 SJR: 0.345 SNIP: 0.46 CiteScore™:: 2.5 H-Index: 67

Indexed in

Regulation of Differentiated Osteoclasts

卷 10, 册 3&4, 2000, 18 pages
DOI: 10.1615/CritRevEukarGeneExpr.v10.i3-4.10
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摘要

Osteoclasts respond to many factors, including endocrines, cytokines, cell-cell interactions, and cell-matrix contacts. For mature osteoclasts, the first level of control occurs through signaling that follows binding to an appropriate substrate. Mononuclear and multinucleate osteoclasts are activated when cell surface integrins, notably but not exclusively αvβ3 integrins, bind to calcified matrices. The binding process results in actin ring formation and deployment of adhesive proteins into a ring shape such that a seal is formed. As this ring forms, components of the ruffled border assemble from diffuse distribution to form the resorption apparatus, which includes the vacuolar-ATPase, carbonic anhydrase, and other key molecules. This review focuses on the control of osteoclast activity, beginning with attachment and ruffled border assembly. Direct and indirect regulation by PTH/PTHrP, genomic and nongenomic effects of estrogen, and gene expression of ruffled border components, carbonic anhydrase, and vacuolar ATPase are reviewed. Finally, the need to understand complex signaling pathway interaction is discussed.

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