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Journal of Long-Term Effects of Medical Implants

年間 4 号発行

ISSN 印刷: 1050-6934

ISSN オンライン: 1940-4379

SJR: 0.184 SNIP: 0.485 CiteScore™:: 1.1 H-Index: 39

Indexed in

Innate Immunity Sensors Participating in Pathophysiology of Joint Diseases: A Brief Overview

巻 24, 発行 4, 2014, pp. 297-317
DOI: 10.1615/JLongTermEffMedImplants.2014010825
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要約

The innate immune system consists of functionally specialized "modules" that are activated in response to a particular set of stimuli via sensors located on the surface or inside the tissue cells. These cells screen tissues for a wide range of exogenous and endogenous danger/damage-induced signals with the aim to reject or tolerate them and maintain tissue integrity. In this line of thinking, inflammation evolved as an adaptive tool for restoring tissue homeostasis. A number of diseases are mediated by a maladaptation of the innate immune response, perpetuating chronic inflammation and tissue damage. Here, we review recent evidence on the cross talk between innate immune sensors and development of rheumatoid arthritis, osteoarthritis, and aseptic loosening of total joint replacements. In relation to the latter topic, there is a growing body of evidence that aseptic loosening and periprosthetic osteolysis results from long-term maladaptation of periprosthetic tissues to the presence of by-products continuously released from an artificial joint.

によって引用された
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  3. Liu Jie, Tang Ling-yun, Wang Yan-gui, Lu Shun-yuan, Zhang En-ning, Wang Zhu-gang, Zhang Hong-xin, Identification of MAVS as a Novel Risk Factor for the Development of Osteoarthritis, Aging and disease, 9, 1, 2018. Crossref

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