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Critical Reviews™ in Immunology

年間 6 号発行

ISSN 印刷: 1040-8401

ISSN オンライン: 2162-6472

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 1.3 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.6 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00079 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.24 SJR: 0.429 SNIP: 0.287 CiteScore™:: 2.7 H-Index: 81

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How Phagocytes Track Down and Respond to Apoptotic Cells

巻 29, 発行 2, 2009, pp. 111-130
DOI: 10.1615/CritRevImmunol.v29.i2.20
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要約

The uptake of apoptotic cells by macrophages and dendritic cells or nonprofessional phagocytes is crucial for development and tissue homeostasis. This is of special importance because deficiencies in the recognition or removal of apoptotic cells may result in autoimmune diseases. The efficient elimination of an apoptotic cell involves contact between the altered cell and the phagocyte, specific recognition, and phagocytosis of the target. These processes are closely associated with the release of anti-inflammatory cytokines and the induction of self-tolerance. This review focuses on the different types of signals, bridging molecules and receptors involved in both steps of the uptake process. Additionally, the role of soluble pattern recognition molecules from the innate immune system, known for a long time to discriminate pathogens from self and more recently to sense altered self, is discussed. This applies to complement Clq, which appears to sense multiple ligands exposed at the apoptotic cell surface, to be involved in their engulfment by phagocytes, and to modulate dendritic cell maturation.

によって引用された
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