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Journal of Environmental Pathology, Toxicology and Oncology

Publicou 4 edições por ano

ISSN Imprimir: 0731-8898

ISSN On-line: 2162-6537

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 2.4 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.8 The Immediacy Index is the average number of times an article is cited in the year it is published. The journal Immediacy Index indicates how quickly articles in a journal are cited. Immediacy Index: 0.5 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00049 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.59 SJR: 0.429 SNIP: 0.507 CiteScore™:: 3.9 H-Index: 49

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Heat Shock Protein 27 Protects against Aminolevulinic Acid-Mediated Photodynamic Therapy-Induced Apoptosis and Necrosis in Human Breast Cancer Cells

Volume 26, Edição 3, 2007, pp. 173-183
DOI: 10.1615/JEnvironPatholToxicolOncol.v26.i3.20
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RESUMO

This study utilized two breast cancer cell lines differing only in their expression of heat shock protein 27 (hsp27). The DB46 cell line was engineered to express high constitutive levels of hsp27, while the DC4 cell line expresses normal low levels of hsp27. The cells were incubated in 1 mM aminolevlinic acid (ALA) 4 hr prior to light exposures (635 nm) ranging from 1 to 20 J/cm2. Both cell lines displayed a dose response to photodynamic therapy (PDT) as assayed by clonogenic survival. LD50s of 2.68 and 1.27 J/cm2 were observed for DB46 and DC4 cells respectively. ALA-PDT-induced resistance to both apoptosis and necrosis in the DB46 cell line was found from TUNEL assays and fluorescence microscopy studies using propidium iodide and Hoechst staining.

CITADO POR
  1. Nokes Brandon, Apel Matt, Jones Carol, Brown Gabrielle, Lang Julie E., Aminolevulinic acid (ALA): photodynamic detection and potential therapeutic applications, Journal of Surgical Research, 181, 2, 2013. Crossref

  2. Broekgaarden Mans, Weijer Ruud, van Gulik Thomas M., Hamblin Michael R., Heger Michal, Tumor cell survival pathways activated by photodynamic therapy: a molecular basis for pharmacological inhibition strategies, Cancer and Metastasis Reviews, 34, 4, 2015. Crossref

  3. Rodríguez Matías Exequiel, Arévalo Daniela Elisa, Milla Sanabria Laura, Cuello Carrión Fernando Darío, Fanelli Mariel Andrea, Rivarola Viviana Alicia, Heat shock protein 27 modulates autophagy and promotes cell survival after photodynamic therapy, Photochemical & Photobiological Sciences, 18, 2, 2019. Crossref

  4. Fan Yipu, Xue Weikang, Schachner Melitta, Zhao Weijiang, Honokiol Eliminates Glioma/Glioblastoma Stem Cell-Like Cells Via JAK-STAT3 Signaling and Inhibits Tumor Progression by Targeting Epidermal Growth Factor Receptor, Cancers, 11, 1, 2018. Crossref

  5. Franchi Leonardo Pereira, de Freitas Lima Jéssica Ellen Barbosa, Piva Henrique Luis, Tedesco Antonio Claudio, The redox function of apurinic/apyrimidinic endonuclease 1 as key modulator in photodynamic therapy, Journal of Photochemistry and Photobiology B: Biology, 211, 2020. Crossref

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