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Journal of Environmental Pathology, Toxicology and Oncology

Published 4 issues per year

ISSN Print: 0731-8898

ISSN Online: 2162-6537

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 2.4 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.8 The Immediacy Index is the average number of times an article is cited in the year it is published. The journal Immediacy Index indicates how quickly articles in a journal are cited. Immediacy Index: 0.5 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00049 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.59 SJR: 0.429 SNIP: 0.507 CiteScore™:: 3.9 H-Index: 49

Indexed in

Toward an Understanding of the Molecular Genetics of Prostate Cancer Progression

Volume 22, Issue 1, 2003, 15 pages
DOI: 10.1615/JEnvPathToxOncol.v22.i1.10
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ABSTRACT

The study of the disease process of prostate cancer has revealed, over many years, numerous chromosomal and genetic alterations associated with the development and progression of this cancer. Although there is much information relating to prostate cancer at the molecular level, little is known as to how these alterations relate to each other. Also, a link between prostate cancer and its likely precursor lesions, such as prostatic intraepithelial neoplasia and atypical adenomatous hyperplasia, is not well established. This review aims to summarize current knowledge of the genetics of prostate cancer and its precursor lesions, with particular mention of the relatively new class of genes involved in the acquisition of the metastatic phenotype, the metastasis suppressor genes.

CITED BY
  1. Konishi Noboru, Shimada Keiji, Ishida Eiwa, Nakamura Mitsutoshi, Molecular pathology of prostate cancer, Pathology International, 55, 9, 2005. Crossref

  2. Sieber Oliver M., Heinimann Karl, Tomlinson Ian P. M., Genomic instability — the engine of tumorigenesis?, Nature Reviews Cancer, 3, 9, 2003. Crossref

  3. Ho Shuk-Mei, Metal Ions as Endocrine Disruptors, in Endocrine Disruptors, 2004. Crossref

  4. Fernández Pedro L., Thomson Timothy M., 3 Alterations of genes and their expression in prostate carcinoma, in Molecular Pathology, Colorectal Carcinoma, and Prostate Carcinoma, 2, 2002. Crossref

  5. Wernert Nicolas, Kaminski Annette, Haddouti El-Mustapha, Hahne Jens Claus, Tumor-Stroma Interactions of Metastatic Prostate Cancer Cell Lines, in Microarrays, 382, 2007. Crossref

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