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Critical Reviews™ in Immunology

Publication de 6  numéros par an

ISSN Imprimer: 1040-8401

ISSN En ligne: 2162-6472

The Impact Factor measures the average number of citations received in a particular year by papers published in the journal during the two preceding years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) IF: 1.3 To calculate the five year Impact Factor, citations are counted in 2017 to the previous five years and divided by the source items published in the previous five years. 2017 Journal Citation Reports (Clarivate Analytics, 2018) 5-Year IF: 2.6 The Eigenfactor score, developed by Jevin West and Carl Bergstrom at the University of Washington, is a rating of the total importance of a scientific journal. Journals are rated according to the number of incoming citations, with citations from highly ranked journals weighted to make a larger contribution to the eigenfactor than those from poorly ranked journals. Eigenfactor: 0.00079 The Journal Citation Indicator (JCI) is a single measurement of the field-normalized citation impact of journals in the Web of Science Core Collection across disciplines. The key words here are that the metric is normalized and cross-disciplinary. JCI: 0.24 SJR: 0.429 SNIP: 0.287 CiteScore™:: 2.7 H-Index: 81

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Regulation of Antiviral CD8 T-Cell Responses

Volume 33, Numéro 6, 2013, pp. 477-488
DOI: 10.1615/CritRevImmunol.2013007909
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RÉSUMÉ

A balanced immune response to a viral pathogen leads to clearance of the virus while limiting immune mediated pathology. Control of this process occurs at all stages of the immune response, including during the induction of an antiviral response, clearance of virally infected cells, and the resolution of this response. Regulation of antiviral immune response is further modified when the immune system fails to clear the pathogen and by the nature of chronic infection itself. A number of processes have been implicated in the regulation of antiviral immune responses, such as the limitation of viral antigen load by interferons, apoptosis through cytokine withdrawal or Fas-mediated killing, and control of these responses by regulatory T cells. This review addresses several of these mechanisms.

CITÉ PAR
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  2. Uyangaa Erdenebileg, Choi Jin Young, Patil Ajit Mahadev, Kim Jin Hyoung, Kim Seong Bum, Kim Koanhoi, Ryu Hyung Won, Oh Sei-Ryang, Eo Seong Kug, Functional restoration of exhausted CD4+ and CD8+ T cells in chronic viral infection by vinegar-processed flos of Daphne genkwa, Comparative Immunology, Microbiology and Infectious Diseases, 39, 2015. Crossref

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  11. Ferraro Diana, De Biasi Sara, Simone Anna Maria, Orlandi Riccardo, Nasi Milena, Vitetta Francesca, Pinti Marcello, Fogliani Marco, Meletti Stefano, Cossarizza Andrea, Sola Patrizia, Modulation of Tregs and iNKT by Fingolimod in Multiple Sclerosis Patients, Cells, 10, 12, 2021. Crossref

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  13. Hoffman Matilde, Chigbu DeGaulle I., Crumley Brenndan L., Sharma Ravi, Pustylnikov Sergey, Crilley Thomas, Ginwala Rashida, Loonawat Ronak, Joseph Julie, Sales Dominic, Wilson Sydney, Jain Pooja, Human Acute and Chronic Viruses: Host-Pathogen Interactions and Therapeutics, in Advanced Concepts in Human Immunology: Prospects for Disease Control, 2020. Crossref

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